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|Title:||Mendelian randomization in inflammatory conditions - the exception rather than the rule?|
|Authors:||Bezzina Wettinger, Stephanie|
Rosendaal, F. R.
Reitsma, P. H.
Felice, Alex E.
|Citation:||Bezzina Wettinger, S., Doggen, C. J. M., Spek, A., Felice, A., Rosendaal, F. R., & Reitsma, P. H. (2014). Mendelian randomization in inflammatory conditions-the exception rather than the rule?. Atherosclerosis, 235(2), e221-e222.|
|Abstract:||Objectives: To assess causality of inflammatory RNA molecules for myocardial infarction (MI) using a Mendelian randomization approach. Methods: Samples were 524 men with a history of MI and 628 controls from the Study of Myocardial Infarction Leiden (SMILE). Macrophage migration inhibitory factor (MIF) G-173C, protein tyrosine phosphatase, non-receptor type, 1 (PTPN1) 1484InsG and chemokine (C-C motif) ligand 3 (CCL3 or MIP1-alpha) C1685T were selected since they are in genes that exhibited elevated mRNA expression measured using multiplex ligation probe-dependent amplification in cases compared to controls.|
|Appears in Collections:||Scholarly Works - FacM&SPB|
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